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Approval summary for erlotinib for treatment of patients with locally advanced or metastatic non-small cell lung cancer after failure of at least one prior chemotherapy regimen.

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The aim of the present work was to examine the effects of a combined treatment of metformin with gefitinib, a selective EGFR-TKI on NSCLC cell lines.

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Evaluation of Three Small Molecular Drugs for Targeted Therapy to Treat Nonsmall Cell Lung Cancer.

EGFR is overexpressed in numerous epithelial and neuroectodermal malignancies, a finding that initially motivated the exploration of EGFR as a novel therapeutic target.

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Radiation Monotherapy Improves Survival in Elderly NSCLC Patients.

The T790M mutation in EGFR kinase causes drug resistance by increasing the affinity for ATP.

Retrospectively observed rash-to-survival correlations suggest that individualized dose titration based on the appearance and severity of skin rash may allow for optimization of egfri therapy and have led to the initiation of “dose-to-rash” trials (increasing the dosage until rash is observed) that are examining dose escalation protocols.

David Ferry, MD, at the European Society for Medical Oncology meeting.

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Most patients (61%) had clinical stage I disease, 31% had stage II disease, and 7% had stage III disease.

Similar findings were also made in the recent human study, where breast tumor concentrations of M523595 represented only about 40% of its plasma concentration, whereas gefitinib showed pronounced (42-fold) tumor penetration ().

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There were no detectable levels of phosphorylated EGFR in mouse lung tumors examined, which is the primary postulated target of these EGFR inhibitors ().

Katayama K, Yoshioka S, Tsukahara S, Mitsuhashi J, Sugimoto Y: Inhibition of the mitogen-activated protein kinase pathway results in the down-regulation of P-glycoprotein.

HER2 is a related gene under study that plays a role in regulating cell growth.

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Immunohistochemical analysis of paraffin-embedded human breast carcinoma using Phospho-PRAS40 (Thr246) (C77D7) Rabbit mAb in the presence of control peptide (left) or antigen specific peptide (right).

Pathologists should determine the adequacy of specimens for EGFR testing by assessing cancer cell content and DNA quantity and quality.

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This finding supports the relationship between tumour response rate, egfri dose, and skin toxicity, thereby substantiating the correlation between egfri activity at the level of the egfr receptor on the skin and in the tumour.

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Radiographically occult lung cancer treated with fibreoptic bronchoscopic electrocautery: a pilot study of a simple and inexpensive technique.

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Such a mechanism would explain both the increased receptor activation after ligand binding and the enhanced inhibition induced by gefitinib.

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Outcomes of stereotactic ablative radiotherapy for central lung tumours: a systematic review.

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More patients in Cohen's study had been fit to receive prior therapy, especially surgery (89 vs 51 %) and chemotherapy (85 vs 63 %), underscoring the fact that more of the patients recruited to our programme had never been fit enough to receive radical treatment.

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Department of Medicine, Harvard Medical School, Boston, Massachusetts, USA.

Eric Logigian, a temporary voting member and professor of neurology at the University of Roch- ester Medical Center.

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Studies have shown that in patients with pulmonary adenocarcinoma who had a base-pair deletion at exon 19 (del746_A750) or a point mutation at exon 21 (L858R), the tumors were highly responsive to EGFR tyrosine kinase inhibitors, and subsequent studies of first-line therapy with these agents showed objective response rates of 54.

This was a significant increase from the percentage of people taking prescription drugs just over a decade earlier.

For this reason, bone marrow, or peripheral blood stem cell harvesting is carried out before the ablative part of the therapy, to enable "rescue" after the treatment has been given.

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The combination of carboplatin and gemcitabine at these doses in this population was well tolerated and active, with a response rate of 37% and median survival of 9 months.

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Despite considerable controversy, it has become an acceptable treatment paradigm and both drugs are approved for maintenance therapy of advanced NSCLC patients in Europe and the USA.

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Preclinical studies support clinical findings implicating T790M as an underlying mechanism of resistance.

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The experimental protocol (158-001) was reviewed and approved by the Institutional Animal Care and Use Committee at Hunan Normal University.

Pooled analysis of the prognostic and predictive effects of KRAS mutation status and KRAS mutation subtype in early-stage resected non-small-cell lung cancer in four trials of adjuvant chemotherapy.

Infrequent side effects, sometimes severe, are interstitial lung pneumonitis and hematuria.

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Food and Drug Administration (FDA) for monotherapy of patients with advanced NSCLC after failure of both platinum-based chemotherapy and docetaxel.

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