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Really helped me with severe anxieties and mood swings. Calms me down, makes me less irritated and obsessive, helps me fall asleep. Better to take it before going to sleep. I tried taking it in the morning and couldn't get through the day (feeling too sleepy and out of it). It works wonders for those with extreme emotions.

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National clinical guidelines in the United States, Canada, and the United Kingdom encourage a variety of approaches to address behavioral symptoms in persons with dementia.

The doctors did not recognize my symptoms as drug-induced and kept prescribing me more medications to treat the adverse reactions, along with labeling me as a psychiatric case and neuro-lyme disease.

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It's mainly used to treat behavior problems in autistic children because it is considerably very powerful in controlling behavior problems compared to other antipsychotics.

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Overall, the clinical use of Formulations A, B, C, or D will eliminate the need for combination oral therapy and reduce time to achieve steady state, with a smaller washout period upon cessation of therapy.

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Brand Names: Risperdal, Risperdal M-Tab What is the most important information I should know about risperidone?

Side-effects include a prolonged in the heart, which can be dangerous for patients with or those taking other drugs that prolong the QT interval.

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For Bipolar Disorder: "I have just started taking this medication and it numbs me to everything.

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Risperidone is used to treat aggression, false perceptions, Tourette's syndrome or behavioral problems in persons with mental retardation.

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SD age of the children at starting risperidone was years.

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As with other drugs that antagonize dopamine D 2 receptors, Risperidone elevates prolactin levels and the elevation persists during chronic administration.

Sustained drug delivery optimizes long-term treatment of patients with schizophrenia.

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The receptor interaction profile of paliperidone is essentially the same as for its parent drug, ie, its action is believed to be mediated mainly through antagonism at dopamine D2 receptors and serotonin 5-HT2A receptors while it has little activity at muscarinic receptors and hence, little potential for anticholinergic side effects while the preponderance for causing hyperprolactinemia can be expected to be the same ().

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Metabolic problems have replaced tardive dyskinesia as an important long-term safety concern in patients receiving maintenance treatment with atypical antipsychotics.

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