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I (78 y.o. male) was diagnosed with NSCLC in March 2011 and in late May I started taking Iressa as a first line of treatment. For the first five months my only side effect was a mild rash. I then had swollen gums and stomach acid. Both quickly responded to antibiotics. 75 days between the March and May CT scans the tumor increased by 86% (from 4.9 x 4 cms). 50 days later in July, after taking Iressa, the CT scan revealed an 83% reduction. From March to July it reduced by 69% and in December, although subtle, there has been further reduction. April 2012. True to form, tumour started growing again after 10 months of Iressa treatment. Still looking healthy, active in garden and taking 4 to 6 miles weekly walks. Only side effect was mild rash mainly on body. Now switching to conventional Chemotherapy today 10th May.
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Iressa is an anticancer drug that slows down the growth of cancer cells. I was prescribed with this medication when my doctor diagnosed me with cancer, three years ago. I have studied much literature about cancer treatment and finally decided to try Iressa as it seemed to be the most beneficial solution with mildest side effects. And I still think that I have made the right choice as I'm still alive what wasn't expected by my doctor! Iressa taught me to fight for my life, successfully!
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it turns out that iressa is an inhibitor of the tyrosine kinase of the egf receptor
and several randomized confirmatory trials failed to demonstrate the benefit of iressa
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Among them, there are endogenous lipid anandamide, tumor necrosis factor, tumor necrosis factor-related apoptosis-inducing ligand, docetaxel, PTX, etoposide, doxorubicin, irinotecan, gefitinib, and semisynthetic retinoid, N-(4-hydroxyphenyl) retinamide (or fenretinide).
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However, it is at present unknown whether blockade of these receptors affects the growth of c-SCLC.
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OK, so perhaps that's a bit of an exaggeration, but Pfizer's CEO Ian Read hasn't been shy about noting the importance of inorganic growth for Pfizer.
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The tyrosine kinase inhibitor ZD1839 (“Iressa”) inhibits HER2-driven signaling and suppresses the growth of HER2-overexpressing tumor cells.
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Lapatinib effectively inhibits EGFR activation, leading to a reduction in STAT1 expression.
If there is still fluid in the lungs, it just means the chemo is useless.
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